XEMA test Peanut


Peanut allergy can present a variety of symptoms, from a mild oral allergy to severe life-threatening systemic reactions, that is, anaphylactic shock or bronchial asthma. Peanut allergy is quite common throughout the world. Peanut-induced anaphylaxis is considered the most fatal among all food allergies. Peanut allergy affects more than 0.5% of children in major populations. The Food Allergen Labeling and Consumer Protection Act identified peanut allergy as one of the top 8 food allergies that must be specifically labelled. In the EU, peanuts are included in the list of allergens established by the European Food Safety Authority, which must be indicated in the food according to EU legislation.


  • Rapid immunochromatographic test for the qualitative determination of peanut antigen in food, kitchens and production facilities.
  • Peanuts belong to legumes, the family of beans or peas.
  • XEMATest Peanut does not detect antigens from grains, legumes, and other nuts.
  • Be sure. Be save.


  • 5 test strips individually packed in sealed bags;
  • 5 sample collection tubes;
  • 1 vial of Sample Extraction Buffer, 22 ml
  • Instructions for use.

In this case, although positive, the peanut-specific IgE is well below the generally accepted positive predictive limit of 14 km / L. I assume that the negative component tests included Ara h 6 as this test is now commercially available. Since there is no history of an acute reaction, it is difficult to interpret the test results. An oral feeding challenge is needed to determine if the child is clinically reactive.

Du Toit and her colleagues identified severe eczema and egg allergy as the strongest predictors of peanut sensitization in babies. They also showed that the duration of eczema was also associated with peanut sensitization. Although peanut-specific IgE was not used in the LEAP intervention, I think it may be helpful to consider that among the 47 babies randomized to the consumption group who had positive peanut skin prick tests and underwent initial peanut test, only 6 had had a positive result of the challenge.

His symptoms were limited to the skin and did not require epinephrine. Koplin et al reviewed the peanut test modalities and proposed an approach that combines peanut-specific IgE with the Ara h 2 test. If sIgE is between 0.35-15 kU / L and Ara h 2 is less than 1.0 km / L, so they recommend considering an oral peanut challenge in the hospital. I have seen mixed results between peanut-specific IgE and peanut component tests. Other allergists have noticed it too. I couldn’t find a post on the frequency of these disparate findings.

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